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By Luigi Amaducci, Marzia Baldereschi (auth.), Robert E. Becker, Ezio Giacobini, Joyce M. Barton, Mona Brown (eds.)

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Extra resources for Alzheimer Disease: From Molecular Biology to Theraphy

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J-P. Michel et al. 1% TABLE IV: Relation of cognitive status at admission and neuropathological diagnosis in 52 autopsied diabetic patients. It appears, therefore, that diabetics presenting with lesions of Alzheimer type display clinically cognitive dysfunction in the form of poor intellectual performance (MMSE<21) in only 43% of cases. DISCUSSION This study is unique insofar as, to our knowledge, it is the first to attempt to establish a correlation between diabetes and dementia based upon neuropathology.

1996): Preclinical evidence of AD in persons homozygous for the E4 allele for apolipoprotein E. New Engl J Med; 334:752-8. Schwartz MW, Figlewicz DP, Baskin DG (1989): Insulin in the brain: a hormonal regulator of energy balance. Mol Neurobiol 3 :71-5. Skoog I, Nilsson L, Palmertz B et al. (1993): A population-based study of dementia in 85-year-olds. New Engl J Med 328:153-8. Small GW, Rosenthal MJ (1992): Coexistence of AD and diabetes mellitus. JAm Geriatr Soc 40: 1075-6. Soininen H, Puranen M, Helkala EL et al.

1992). , 1992). Non-demented cases have been described with NFT preferentially distributed in the entorhinal cortex, and lower NFT counts in the CA 1 and temporal neocortex, and numerous SP in the neocortex (Braak and Braak, 1991). Also, Braak and Braak (1990, 1991) have demonstrated that the transition zone between the allo- and neocortex was consistently involved 26 C. Bouras et al. early in the degenerating process, and have proposed a neuropathologic staging of dementia based on the progressive alteration of the hippocampal formation.

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